GSE100328

KMT2C medaites the estrogen dependence of breast cancer through regulation of ERα enhancer function

KMT2C is a key regulator of ERα activity whose loss allows for hormone independent proliferation in MCF7 cells

Provenance — who produced it, who reused it

Linked to 1 papers in the literature. Roles are inferred factual signals (who deposited the data vs who reused it), with counts — never a judgement about any author.

Standardized, field-standard QC computed by touching the data — every metric states how it was obtained

measured = computed from the data · extrapolated/reported = derived or from the repository · dq-1.0 · provisional — verify independently