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Pattern occupation of HDACs on chromatin determines PI3K/AKT inhibitor resistance in cancers [ChIP-seq]
Blocking BRD4 or HDACs induces paradoxically similar gene expression, comparably attenuates PI3K/AKT inhibitor resistance in multiple cancer cells.
Provenance — who produced it, who reused it
Linked to 1 papers in the literature. Roles are inferred factual signals (who deposited the data vs who reused it), with counts — never a judgement about any author.
Deposited / produced by
Haojie HuangDi WuZhenqing Ye
Reused by
- UALCAN: An update to the integrated cancer data analysis platfor... 2022 · 2,521 cites
Deep data QC
metadata only · no data-level QC for this typeStandardized, field-standard QC computed by touching the data — every metric states how it was obtained
Data type / assay
ChIP-seq
Organism
Homo sapiens
Instrument
Illumina HiSeq 3000
Platform
ILLUMINA
Read type
short-read
Files available
FASTQ (raw reads)
N numbers (samples, groups)
18 / 18 runs
Completeness
100%
Metrics (value · how obtained)
checksum ok
yes
reported
total bases
33297576048
reported
total reads
326446824
reported
supplementary file types
BW
reported
QC cost
22 s compute
measured = computed from the data · extrapolated/reported = derived or from the repository · dq-1.0 · provisional — verify independently