Experiments
Searchable full-text extractions: founding hypothesis, core claims, experimental setups, key results and statistics — pulled out of each paper as structure. Search a cell line, an assay or an entity (e.g. HUH7) and find every paper that worked with it. This corpus stands on its own: most entries carry no reproduction assessment (yet).
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Variation in the E2-binding domain of HPV 16 is associated with high-grade squamous intraepithelial lesions of the cervix.
PMID 11308254 · PMC2363853 · British journal of cancer · 2001 · 8 claims · 6 setups
E2 gene disruption in the analysed regions is significantly more frequent in high-grade SILs than low-grade SILs
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The role of p53 inactivation in human cervical cell carcinoma development.
PMID 7841033 · PMC2033612 · British journal of cancer · 1995 · 8 claims · 7 setups
HPV DNA sequences were detected in 43 of 47 (91.5%) primary uterine cervical cancers
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11q13 amplification status and human papillomavirus in relation to p16 expression defines two distinct etiologies of head and neck tumours.
PMID 17003776 · PMC2360598 · British journal of cancer · 2006 · 8 claims · 7 setups
HPV-positive HNSCC tumours are significantly less likely to carry 11q13 amplification than HPV-negative tumours
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Elderly Japanese women with cervical carcinoma show higher proportions of both intermediate-risk human papillomavirus types and p53 mutations.
PMID 10098748 · PMC2362249 · British journal of cancer · 1999 · 8 claims · 3 setups
HPV DNA was detected in 44 of 46 (96%) cervical carcinomas from elderly women
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Integrative genomics analysis of chromosome 5p gain in cervical cancer reveals target over-expressed genes, including Drosha.
PMID 18559093 · PMC2440550 · Molecular cancer · 2008 · 7 claims · 6 setups
Gain of chromosome 5p is the most frequent genomic alteration in invasive cervical cancer