Experiments
Searchable full-text extractions: founding hypothesis, core claims, experimental setups, key results and statistics — pulled out of each paper as structure. Search a cell line, an assay or an entity (e.g. HUH7) and find every paper that worked with it. This corpus stands on its own: most entries carry no reproduction assessment (yet).
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Has reproduction
Genome scans of facial features in East Africans and cross-population comparisons reveal novel associations.
PMID 34411106 · PMC8375984 · PLoS genetics · 2021 · 8 claims · 5 setups
Genome scans of 3D facial shape phenotypes in Tanzanian children identified 20 loci significant at p < 2.5 × 10^-8
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Genetic subtraction reveals divergent pathways and targets in anxiety-related and anxiety-independent TMD.
PMID 41796324 · PMC13081294 · The journal of headache and pain · 2026 · 8 claims · 8 setups
Anxiety shows significant genetic correlation with TMD (rg=0.4417, p=1.98x10^-19)
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Discovery and characterization of gene-by-environment and epistatic genetic effects in a vertebrate model.
PMID 41672067 · PMC13174225 · Cell genomics · 2026 · 7 claims · 7 setups
A segregation analysis in an F2 medaka cross identified 16 QTLs linked to embryonic heart rate variation across temperatures
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Large-scale multi-omics unveils host-microbiome interactions driving root development and nitrogen acquisition.
PMID 41634153 · PMC12929062 · Nature plants · 2026 · 8 claims · 8 setups
Multi-omics integration of root transcriptome, rhizosphere 16S microbiome and root/shoot ionome across 175 B. napus accessions at two field sites links host gene expression to microbiome assembly and nutrient traits
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Enhancing KLF15 activity in cardiomyocytes: a novel approach to prevent pathological reprogramming and fibrosis via nuclease-deficient dCas9VPR.
PMID 41771837 · PMC12953643 · Signal transduction and targeted therapy · 2026 · 8 claims · 8 setups
KLF15 transcriptional activity shows the most significant change among TFs in pathological cardiomyocytes during progressive hypertrophic remodeling, reflecting less effective repression of disease-associated genes.