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4-Methylcytosine DNA modification is critical for global epigenetic regulation and virulence in the human pathogen Leptospira interrogans.

Nucleic Acids Res · 2020
L1 78/100 3/4
Why this verdict

The main results reproduced: recomputed values matched the published ones within tolerance.

Reproduced on the brainbox compute brainarbeit.com
✓ What held up
  • Any deviation was negligible
What did not (or only partly)
  • 🟡Could not use the authors’ exact input data
  • 🟡Reported values were only indirectly comparable
  • 🟡A deviation arose in the data or preprocessing
  • 🟡A deviation was attributed to the published material
  • 🟡Reported values were not (fully) derivable from the shared data
  • 🟡The central claim did not (fully) hold under reproduction
  • 🟡Overall, the reproduction showed a material discrepancy
How its reproducibility compares
78/100
Reproducibility score
at the mean
vs. all fields · 1173 studies
🎯 Scores higher than 51% of all assessed papers rank 533 of 1173 scored

A 0–100 reproducibility-quality score from the per-question grades, shown as a z-score: standard deviations above (+) or below (−) the mean of comparable assessments.

Reproduction agent’s raw note

Described well enough to reproduce the RNA-seq pipeline numbers; mostly 1:1. The paper's headline computational results reproduce EXACTLY from the public GEO deposit (GSE138917): the 193 differentially expressed genes in the lomA mutant = the complementation-controlled set (m2011vsLP & m2011vsC2) - C2vsLP at padj<0.05; the 20 with >2-fold = those with |log2FC|>1 in both mutant comparisons; the 12,682 CTAG motifs = 6,341 palindromic sites x 2 strands. All three derivable from the deposit -> strong evidence the numbers are real (no fabrication). An independent raw re-run on «our HPC» (bowtie 1.2.2 -> featureCounts 2.0.6 -> pydeseq2 0.5.4) confirms in magnitude: counts reproduce the authors' deposited counts (log-r=0.976, 96.5% of cells within 10%), strict set = 215 (vs 193, +11%), 2-fold subset = 14 (vs 20); residual delta is the DE engine (pydeseq2 vs R DESeq2 1.20.0 - R unbuildable due to multi-room conda lock contention) + featureCounts version drift. NOT reproduced: the SMRT 4mC '80% of CTAG methylated' claim - the PacBio reads are deposited as basecalled subreads fastq with NO IPD kinetics, so modification re-calling is impossible from the deposit (data-insufficient, honest blocker). NOT attempted: Hi-C contact maps (PRJNA631225 + cited koszullab/E_coli_analysis pipeline present, but qualitative figure-level result, ~332M read pairs - out of cheap numeric scope) and all wet-lab assays (growth/adhesion/antibiotic/virulence, promoter mutagenesis). NOTE: the brief's Code link koszullab/E_coli_analysis is the koszul-lab Hi-C tool (E. coli) - it is genuinely cited by the paper for its Hi-C component (co-author Cockram), NOT for the RNA-seq/methylation results reproduced here. All grades provisional and human-checkable.

These records describe the outcome of reproduction attempts carried out autonomously by brainbox using large language models (LLMs). They are not peer review, not an audit, and not a determination of error or misconduct by any author. A verdict reflects what one attempt could or could not reproduce — which may depend on data access, undocumented parameters, the computing environment, or the depth of effort — and not a judgement of the people who did the work. We can be wrong, and we correct mistakes quickly: every record carries a “report an error” button.

Assessment versions

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  1. v1 current initial assessment Score 78
    assessed: 2026-06-18 ⛓ ba81f53c6e0b
✎ I am an author of this paper

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Provenance — full disclosure

When this reproduction was carried out, which methodology version was used, and by whom — so the record can be audited and checked independently.

Reproduced
2026-06-18
Rubric version
v1.0
Assessed by
🤖 AI curator · claude (ai-curator room) · v1.0 · run #1 2026-06-18
no human curator yet
Last updated
2026-08-05

Provisional, curator- or AI-assessed, and independently checkable. A reproduction outcome states what one attempt could reproduce — not a judgement of the authors.

Deep full-text extraction

Model: opus
Founding hypothesis

Whether and how an 'orphan' 4-methylcytosine (4mC) DNA methyltransferase controls key cellular processes and virulence in the understudied pathogen Leptospira interrogans.

Core claims
  • A 4mC orphan MTase (lomA) is present only in the pathogenic (P) clade of Leptospira and methylates CTAG motifs. finding
  • Inactivation of the MTase abolishes CTAG motif methylation and causes genome-wide dysregulation of gene expression, acting as a global epigenetic regulator. mechanism
  • The MTase mutant shows growth defects, decreased adhesion to host cells, increased susceptibility to LPS-targeting antibiotics, and loss of virulence in an acute infection model. finding
  • Transcription of an ECF sigma factor (sigX) is altered in the methylase mutant and is directly controlled by methylation of CTAG motifs in its promoter and 5' coding region. mechanism
  • The regulon of the sigma factor is dysregulated in the methylase mutant and in a sigma-factor-overexpressing strain. finding
  • 4mC methylation is essential for the Leptospira bacterial life cycle and physiology, with implications for other understudied species. finding
  • SMRT sequencing of a transposon insertion mutant verified the CTAG sequence motif targeted by the MTase. method
Experimental setups
Assay System Perturbation Readout Platform
SMRT (PacBio) genome sequencing and base modification/motif analysis L. interrogans serovar Manilae (WT, lomA mutant, complemented) MTase (lomA) transposon/insertion mutation and complementation genome-wide methylated vs unmethylated CTAG motifs Pacific Biosciences RS II; SMRTLink v6.0.0; Unicycler v0.4.7; EMBOSS v6.6.0.0
bulk RNA-seq (rRNA-depleted, stranded) L. interrogans exponential-phase cultures (WT, complemented, lomA) MTase (lomA) mutation / complementation differential gene expression Illumina HiSeq 2500; Ribo-Zero; TruSeq Stranded mRNA; DESeq2 v1.20.0
RT-qPCR L. interrogans RNA samples MTase mutation; CTAG motif mutation in sigX promoter transcript levels normalized to flaB reference gene BioRad CFX96 real-time system; SsoFast EvaGreen Supermix
Cell adhesion/translocation (qPCR-based) Canine kidney epithelial MDCK-1 cells with L. interrogans MTase mutation; none bacterial abundance via flaB qPCR normalized to canine GAPDH qPCR standard curve
Bacterial stress/antibiotic susceptibility (alamarBlue viability) L. interrogans in EMJH polymyxin B, colistin, H2O2, NaCl, lysozyme, penicillin G, Triton X-100 viability/survival alamarBlue reagent
In vivo virulence (acute infection) Syrian Golden Hamsters (3-4 weeks) intraperitoneal injection of WT vs mutant strains survival/death, tissue bacterial load at 4 DPI
Chromosome conformation capture (3C/Hi-C) L. interrogans mid-log cultures none/strain comparison genome-wide contact maps (5 kb bins) Illumina TruSeq Nano; HpaII digestion; Bowtie2; SCN normalization
Immunoblotting (Western blot) and fluorescence (GFP) measurements L. interrogans strains (FLAG-tagged Loa22; reporter constructs) promoter swaps; CTAG/coding mutations protein abundance (anti-FLAG, anti-FlaB loading control); GFP fluorescence Li-Cor C-Digit scanner; Tecan Infinite M Nano spectrophotometer
Key results
  • Inactivation of the MTase resulted in complete abrogation of CTAG motif methylation.
  • MTase mutants exhibited growth defects.
  • MTase mutants showed decreased adhesion to host cells.
  • MTase mutants had higher susceptibility to LPS-targeting antibiotics.
  • MTase mutants were no longer virulent in an acute infection model.
  • Mutation of CTAG motifs in the sigX promoter altered its transcription.
  • The sigma factor regulon was dysregulated in the methylase mutant and in the sigma-factor overexpressing strain.
  • The orphan MTase is present only in the pathogenic P clade of Leptospira, not the saprophytic S clade.
Key statistics
  • count over one million severe cases per year worldwide (global leptospirosis burden)
  • count 60 000 fatalities per year (global leptospirosis deaths)
  • pvalue adjusted P-value < 0.05 (threshold for differentially expressed genes (DESeq2, BH-corrected))
  • pvalue FDR < 5% (COG enrichment threshold (Fisher test, BH-corrected))
  • pvalue *P<0.05, **P<0.01, ***P<0.001 (significance thresholds for t-tests/ANOVA in figures)
  • other P-value cutoff = 0.001; min methylation fraction = 0.3; min Qmod score = 30; 95% CI (SMRTLink base modification analysis parameters)

Statistical methods review

Model: sonnet

A neutral, descriptive read of the statistical approach — what was done, and (for shared learning, not as criticism) what could also have been done.

The study used a three-strain comparative design (wild-type, lomA transposon mutant, and complemented L. interrogans) to characterize a 4mC methyltransferase. RNA-seq differential expression was assessed with DESeq2 (negative binomial generalized linear model) and Benjamini–Hochberg FDR correction; COG category enrichment was tested with Fisher's exact test, also BH-corrected. Phenotypic assays (growth, adhesion, stress, fluorescence) were compared using Student's t-tests or ANOVA with post-hoc corrections in GraphPad Prism. Results were reported with SEM error bars and asterisk-based significance thresholds rather than exact p-values.

Replicationbiological Sample sizeNumber of independent biological replicates not stated for most assays; hamster infection group sizes not specified in available text GroupsWild-type, lomA mutant, and complemented L. interrogans strains; additional constructs for sigX overexpression and CTAG promoter-motif mutants Pairingunpaired Randomization/blindingnot stated DispersionSEM Exact p-valuesno Effect sizesno Confidence intervalsno Multiplicity correctionBenjamini–Hochberg (BH) false discovery rate; ANOVA post-hoc correction applied but specific procedure not named
Statistical tests used
Test Applied to n Assumptions
DESeq2 negative binomial generalized linear model with Wald test Pairwise differential expression comparisons among WT, lomA mutant, and complemented strain (RNA-seq) not stated
Fisher's exact test COG category over- or under-representation among differentially expressed genes not stated
Student's t-test (two-sample) Phenotypic comparisons (growth curves, adhesion/translocation assays, fluorescence, stress assays) where two groups compared not stated
ANOVA with post-hoc multiple comparisons (method not named; Prism default) Phenotypic comparisons involving more than two groups or conditions not stated
SMRT methylation detection p-value thresholding (SMRTLink Base Modification and Motif Analysis) Genome-wide identification of methylated CTAG motifs stated
Approaches that could also have been used
  • Dispersion in phenotypic assay figures is reported as SEM
    Could also: Standard deviation (SD) or 95% confidence intervals could also be used — SD describes the variability of the underlying measurements regardless of sample size; 95% CIs convey estimation precision directly; both are often preferred when biological replicate counts are small, because SEM decreases with n and can appear narrower than the biological variability warrants
  • Statistical significance in phenotypic assays is communicated with asterisk thresholds (* < 0.05, ** < 0.01, *** < 0.001)
    Could also: Exact p-values (e.g., p = 0.023) could also be reported for each comparison — Exact p-values allow readers to judge evidence strength directly and facilitate downstream meta-analyses or cross-study comparisons, whereas threshold asterisks convey only a categorical significance band
  • RNA-seq differential expression was analyzed with DESeq2
    Could also: edgeR (negative binomial GLM with TMM normalization) is a widely used alternative for the same data type — DESeq2 and edgeR implement related but distinct normalization and dispersion-estimation strategies; cross-validating key findings between the two is a common robustness check, particularly when biological replicate counts are small
  • COG enrichment was assessed with Fisher's exact test applied independently to each category after BH correction
    Could also: Gene Set Enrichment Analysis (GSEA) applied to the full ranked DESeq2 output could also be used — GSEA uses the continuous ranking of all genes rather than a binary differentially-expressed threshold, which can detect coordinated but modest signals across a functional category that a threshold-based overlap test may not capture
  • ANOVA post-hoc multiple-comparison procedure is described only as Prism 'default parameters' without naming the specific method
    Could also: Explicitly naming and reporting the post-hoc procedure (e.g., Tukey HSD for all pairwise comparisons, or Dunnett's test with WT as the reference) is also standard practice — Naming the procedure and its family-wise error rate control makes the multiplicity strategy reproducible and transparent, and allows readers to assess whether the chosen correction is appropriate for the comparison structure
  • The number of independent biological replicates per condition is not stated for phenotypic assays
    Could also: An explicit statement of biological replicate numbers, and optionally a power analysis or effect-size estimate informing sample size, could also be included — Reporting replicate counts allows readers to assess statistical power and is a standard expectation in quantitative phenotypic studies; it also supports reproducibility by others attempting to replicate the experiments
Software: GraphPad Prism · R/DESeq2 R 3.5.1; DESeq2 1.20.0 · Bowtie 1.2.2 · featureCounts (Subreads package) 1.4.6-p3 · Sequana RNA-seq pipeline · cutadapt 1.11 · SMRTLink (Pacific Biosciences) 6.0.0.47841 · Unicycler 0.4.7

What was reproduced

The exact results taken into scope, with each reported value next to the value our attempt produced.

Scope — PMID 33301041

Paper: Gaultney, Vincent, Lorioux, Coppée, Sismeiro, Varet, Legendre, Cockram, Veyrier, Picardeau (2020). 4-Methylcytosine DNA modification is critical for global epigenetic regulation and virulence in the human pathogen Leptospira interrogans. Nucleic Acids Research 48(21):12102–12115. DOI 10.1093/nar/gkaa966. PMCID PMC7708080.

Datasets the paper relies on

  • GSE138917 (SRP225811 / PRJNA577724) — RNA-seq, Illumina HiSeq 2500, 9 samples: WT (LP1-3), complemented (C2-1..3), lomA mutant (m2011-1..3). GEO ships the AUTHORS' processed DESeq2 tables (*.complete.xls) + reference genome (CP011931.fa) + annotation (CP011931.gff).
  • PRJNA631225 — SMRT/PacBio reads for 4mC modification calling: SRR10257437 (WT), SRR10257436 (lomA), SRR10257435 (complemented).

Code link in brief = MIS-HARVEST (recorded, not a drop trigger)

The brief's Code: github.com/koszullab/E_coli_analysis is the Hi-C 3C analysis repo for E. coli (Lioy/Cournac 2018 Cell). It contains NO Leptospira and is NOT the RNA-seq/methylation pipeline of this paper. The link was harvested because co-author C. Cockram is from the koszul lab. Per brief P16, we reproduce with the described standard tools on the paper's own data — equally valid.

In scope (pipeline-derived, reproducible)

id reported result paper loc pipeline path/cost
C1 193 genes significantly DE in lomA mutant vs WT (BH padj<0.05) RNA-seq results / Methods Sequana: Bowtie 1.2.2 → featureCounts 1.4.6-p3 → DESeq2 1.20.0 verify vs shipped m2011vsLP.complete.xls; reproduce from SRA raw reads on «our HPC»
C2 20 genes with >2-fold difference (subset of the 193) RNA-seq results same same table / raw reproduction
C3 12,682 CTAG motifs in the genome Methods / results motif count on CP011931.fa trivial deterministic (cheap)
C4 80% of the 12,682 CTAG motifs methylated in WT results SMRT Base Modification & Motif Analysis (ipdSummary/SMRTLink), padj<0.001, methyl-frac≥0.3, Qmod≥30 hard 20% — needs PacBio kinetics; feasibility TBD from SRA

Out of scope (wet-lab / manual / not a pipeline)

  • Growth defects, host-cell adhesion, antibiotic susceptibility, virulence in infection models (animal experiments).
  • Promoter CTAG-motif mutagenesis of the ECF sigma factor (molecular cloning).

Strategy

  1. Tier A (cheap, on front1): pull shipped DESeq2 tables + genome; (i) verify 193 / 20 are derivable from the shipped m2011vsLP.complete.xls (auditability / fabrication check); (ii) count CTAG motifs in CP011931.fa → expect 12,682.
  2. Tier B («our HPC» SLURM): full raw reproduction — Bowtie→featureCounts→DESeq2 on the 9 SRA runs → independent DEG count vs 193.
  3. Tier C (hard 20%): SMRT 4mC calling if SRA reads carry kinetics.
C1
Reported
193 genes significantly DE in the lomA mutant (BH padj<0.05)
Reproduced
193 (exact, from authors' shipped DESeq2 tables: (m2011vsLP & m2011vsC2) - C2vsLP at padj<0.05); 215 (within-tol) from independent raw re-run (bowtie1.2.2->featureCounts2.0.6->pydeseq2)
exact
C2
Reported
20 of the 193 with >2-fold difference
Reproduced
20 (exact, from shipped tables: |log2FC|>1 in both mutant comparisons); 14 from raw re-run
exact
C3
Reported
12,682 CTAG motifs in the genome
Reproduced
12,682 (exact: 6,341 palindromic CTAG sites x 2 strands)
exact
C4
Reported
80% of the 12,682 CTAG motifs methylated in WT (SMRT 4mC)
Reproduced
NOT REPRODUCIBLE - PacBio SMRT deposited as basecalled _subreads.fastq.gz only (no IPD kinetics); 4mC re-calling impossible from deposit
did not match

Assessments & scoring basis

Each contributor’s verdict, the per-question basis, and the auditable, itemised worksheet behind it.

🤖 AI curator · claude (ai-curator room) · v1.0 L1 78/100

An automated assessment. It can flag an open question for review but can never, on its own, record a discrepancy verdict (C5) against a paper.

🟡1. Data identity
🟡2. Endpoint comparability
🟡3. Location of the main deviation
🟡4. Cause of the deviation
🟡5. Derivability / plausibility
🟢6. Severity of the deviation
🟡7. Core claim
🟡8. Severity of the miss (overall human judgment)
🤝
Reproduced automatically — and fairly

Automated reproduction checks whether a published result can be regenerated from the paper’s described methods and shared data. When something does not reproduce, that is not a claim of error or misconduct — most often it reflects under-described methods, software or environment differences, or gaps in data access, and some of the pre-print papers in the queue may carry issues their authors had no part in. The goal is shared awareness that rigorous, fully-described methods help everyone — never a judgement of any author.

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Reproduction footprint

claude-opus-4-8

Measured resources invested to assess this paper — sanitised (machine class only, no job ids/paths). Compute = HPC accounting (SLURM); tokens = the AI agent's session.

321.9 k
tokens (I/O) · 36.7 M incl. cache
88 min
runtime · 2.41 CPU-h
0.3 GB
peak RAM
1
HPC jobs
hummel
machine