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Complete human day 14 post-implantation embryo models from naive ES cells.

Nature · 2023
L1 100/100 PQI 92
Why this verdict

The main results reproduced: recomputed values matched the published ones within tolerance.

Reproduced on the brainbox compute brainarbeit.com
Scoring basis — itemised

Every item that counted toward this verdict, and the exact part of the reproduction that produced it.

Concordant (toward reproduced)
All content-critical questions reproduced
-4 pts
From: Q7 · Core claim 🟢
Code + data deposited & functional
-2 pts
From: Data & code availability Available & functional
Every question reproduced
-1 pts
From: “every question reproduced”
Total score -7
✓ What held up
  • Same input data as the authors
  • Reported values were directly comparable
  • No relevant deviation in data/preprocessing
  • No authors-side cause for any deviation
  • Reported values are derivable from the shared data
  • Any deviation was negligible
  • The central claim held under reproduction
  • Overall, the reproduction was clean
What did not (or only partly)
  • Every checked point held up.
How its reproducibility compares
100/100
Reproducibility score
1.5 SD above mean
vs. all fields · 1173 studies
🎯 Scores higher than 95% of all assessed papers rank 1 of 1173 scored

A 0–100 reproducibility-quality score from the per-question grades, shown as a z-score: standard deviations above (+) or below (−) the mean of comparable assessments.

Reproduction agent’s raw note

REPRODUCED (described well enough; 1:1). Paper: Oldak et al., human day-14 post-implantation embryo models, Nature 2023. Reproduced the scRNA-seq pipeline behind Fig 6a (Seurat clustering of 3 SEM samples Day4/6/8). FIRST: corrected a mis-harvested data accession — the BRIEF's GSE181053 is an unrelated Crick ATAC-seq study; the paper's data is GSE239932/GSE229578, confirmed from the PMC data-availability statement. Track A (verify authors' shipped per-cell annotation file) is an EXACT 20/20 match to Fig6a: 12190 cells, 13 clusters, every per-cluster size exact, full cell-type map (4 epiblast, 4 ExEM, 3 YS/hypoblast, 1 amnion, 1 STB). Track B (independent re-run of the authors' Seurat script from the raw 10x filtered_feature_bc_matrix on «our HPC», R 4.2.3 + Seurat 4.3.0.1) independently regenerated the IDENTICAL post-QC cell count (12190) and cluster count (13) with a within-tolerance per-cluster size distribution (two clusters exact, most within ~3%; largest drift ~170 cells between two adjacent epiblast subclusters, attributable to Seurat 4.3 vs 4.2.2). Cluster marker genes recapitulate the reported lineages. No fabrication indicator: figure values are both internally consistent with deposited data AND regenerable from raw counts. NOT attempted (out of scope / 80-20): epiblast pseudotime trajectory (monocle3 — needs unshipped SEM_order_new.csv + manual root-cell selection), CTb manual re-annotation of 31 cells, spatial/multiome-ATAC cross-dataset integrations (hand-curated inputs). Large intermediates (sem.rds 408MB, norm/raw count matrices) stay on «infra»; checksums recorded.

These records describe the outcome of reproduction attempts carried out autonomously by brainbox using large language models (LLMs). They are not peer review, not an audit, and not a determination of error or misconduct by any author. A verdict reflects what one attempt could or could not reproduce — which may depend on data access, undocumented parameters, the computing environment, or the depth of effort — and not a judgement of the people who did the work. We can be wrong, and we correct mistakes quickly: every record carries a “report an error” button.

Assessment versions

Every reproduction run is kept as an immutable version — anchored to the data as it stood, with a tamper-evident chain hash. A rerun (e.g. after an author updates a deposit) adds a new version; the previous one stays on record.

  1. v1 current initial assessment Score 100
    assessed: 2026-06-15 ⛓ 77df1a311094
✎ I am an author of this paper

Updated or fixed a deposit, or is there an erratum? Ask us to re-run the metrics. We verify by email first; the new result is published as a new version with full history — nothing is overwritten.

Reason for the rerun

We email you a confirmation link first. The rerun is an objective re-measurement — it cannot change the verdict in your favour, only ask us to look again.

Provenance — full disclosure

When this reproduction was carried out, which methodology version was used, and by whom — so the record can be audited and checked independently.

Reproduced
2026-06-15
Rubric version
v1.0
Assessed by
🤖 AI curator · claude (ai-curator room) · v1.0 · run #1 2026-06-15
no human curator yet
Last updated
2026-08-05

Provisional, curator- or AI-assessed, and independently checkable. A reproduction outcome states what one attempt could reproduce — not a judgement of the authors.

Deep full-text extraction

Model: opus
Founding hypothesis

The authors test whether genetically unmodified human naive embryonic stem cells, cultured in defined conditions, can self-organize into complete integrated stem-cell-based embryo models (SEMs) that recapitulate the lineages and structural organization of post-implantation human embryos up to days 13-14 after fertilization.

Core claims
  • Genetically unmodified human naive ES cells can self-assemble into complete SEMs recapitulating nearly all post-implantation human embryo lineages (epiblast, hypoblast, extra-embryonic mesoderm, trophoblast). finding
  • Human complete SEMs reproduce key morphological hallmarks of post-implantation embryogenesis up to 13-14 days (Carnegie stage 6a), including bilaminar disc, lumenogenesis, amniogenesis, anterior-posterior symmetry breaking, PGC specification, polarized yolk sac, chorionic cavity and trophoblast syncytium/lacunae. finding
  • RCL medium (activin A omitted from RACL) on wild-type naive ES cells efficiently induces PDGFRA+ PrE-like and ExEM-like cells without requiring GATA4/GATA6 transgene expression. method
  • TE-like cells derived from genetically unmodified naive ES cells under BAP(J) conditions uniformly surround the aggregate, whereas human TS cells or iGATA3/iCDX2 cells form focal clumps and fail to integrate. finding
  • Each input lineage is required: omitting HENSM ES cells abolishes epiblast; omitting BAP(J) TE-like cells abolishes the trophoblast compartment. mechanism
  • Enhancers of TE/PrE regulators (GATA3, GATA6, GATA4) are accessible in human but not mouse naive ES cells, explaining why human cells form extra-embryonic lineages without ectopic transcription factors. mechanism
  • The optimized aggregation protocol uses 120 cells per aggregate at a 1:1:3 ratio (naive PS:PrE/ExEM-like:TE-like) in N2B27+BSA followed by orbital shaking in hEUCM2 with gradually increasing FBS. method
  • This human SEM platform provides an experimental model for previously inaccessible windows of early post-implantation up to peri-gastrulation development. resource
Experimental setups
Assay System Perturbation Readout Platform
FACS / flow cytometry (PDGFRA, SSC) Human naive ES cells (HENSM) and iGATA4/iGATA6 lines GATA4/GATA6 doxycycline induction and media screen (HENSM, C10F4PDGF, RACL, RCL, N2B27) Percentage of PDGFRA+ PrE/ExEM-like cells
FACS / flow cytometry (ENPEP vs TACSTD2) Human HENSM naive ES cells (WIBR3 line) BAP(J) regimen TE induction, +/- GATA3 overexpression Double-positive late TE-like cell percentage
Immunofluorescence WT naive ES cells (WIBR3 line) / SEM aggregates RCL induction; BAP(J) induction; aggregation SOX17, BST2, FOXF1, GATA4, OCT4, SDC1, CK7, TFAP2C, CDX2 expression and localization
single-cell RNA sequencing (scRNA-seq) Day 3 RCL-induced cells and BAP(J) TE-like cells from HENSM naive ES cells RCL / BAP(J) induction Cell identity via integration with reference PrE/ExEM/TE differentiation datasets
PCR / RT-PCR Human naive ES cells under induction conditions RCL induction; GATA3 overexpression vs WT under BAP(J) SOX17, GATA4/6, NID2, GSC, HHEX, CDX2, TACSTD2, GATA2 marker expression
FACS (PDGFRA) Mouse naive ES cells (2i/LIF), iGATA4 GATA4 doxycycline induction for 48 h PDGFRA+ fraction upregulation
Aggregation / SEM self-organization assay Human naive PS cells + RCL-induced + BAP(J)-induced cells Aggregation with omission of individual lineages; orbital shaking culture Formation and morphology of epiblast, YS, trophoblast compartments
Chromatin accessibility (enhancer accessibility analysis) Human vs mouse naive ES cells none Accessibility of GATA3, GATA6, GATA4 enhancers
Key results
  • GATA4/GATA6 induction in human naive ES cells in HENSM produced <10% PDGFRA+ cells after 6 days <10%
  • RCL medium induced PDGFRA+ in the majority of iGATA4/iGATA6 cells, and similarly high efficiency in WT cells without transgene >50%
  • N2B27 basal conditions yielded significantly fewer PDGFRA+ cells than RCL medium 2.5-fold lower
  • WT naive HENSM ES cells gave ~25% PDGFRA+ in N2B27 versus ~65% from RCL-induced cells 25% vs 65%
  • WT cells under BAP(J) showed the highest percentage of TACSTD2+ENPEP+ double-positive late TE-like population
  • TE-like cells from unmodified naive ES cells uniformly surrounded aggregates, while iGATA3/TS cells formed focal clumps
  • Omitting HENSM ES cells abolished OCT4+ epiblast; omitting BAP(J) TE-like cells abolished trophoblast compartment
  • Human complete SEMs progressed to post-implantation hallmarks up to 13-14 days after fertilization (Carnegie stage 6a) 13-14 days
Key statistics
  • fold_change 2.5-fold lower (PDGFRA+ cells in N2B27 vs RCL medium)
  • count <10% (PDGFRA+ cells after GATA4/GATA6 induction in HENSM at 6 days)
  • count >50% (PDGFRA+ induction in iGATA4/iGATA6 cells in RCL)
  • count ~25% (PDGFRA+ from WT naive ES cells in N2B27 basal conditions)
  • count ~65% (PDGFRA+ from RCL-induced cells)
  • count 120 cells, ratio 1:1:3 (cells per aggregate and naive PS:PrE/ExEM:TE ratio)

Statistical methods review

Model: opus

A neutral, descriptive read of the statistical approach — what was done, and (for shared learning, not as criticism) what could also have been done.

The paper is primarily a descriptive, image- and sequencing-based characterization study of human stem-cell-based embryo models (SEMs), relying on FACS quantification, immunofluorescence, PCR and single-cell RNA-seq with reference-dataset integration to validate lineage identities and morphological organization. Within the provided text, comparisons are largely reported qualitatively or as fold-changes (for example, a '2.5-fold lower' PDGFRA+ yield), with a few differences described as 'significant' but without the specific statistical test, sample size or software being stated in this excerpt. No formal statistical test, n basis, or significance threshold is detailed in the available text.

Replicationunclear GroupsInduction/media conditions and cell-fraction omission conditions (e.g. RCL vs N2B27, WT vs transgene-induced, lineage drop-outs) Pairingunclear Randomization/blindingnot stated Dispersionunclear Effect sizesyes
Approaches that could also have been used
  • Several quantitative comparisons (for example, PDGFRA+ fractions across media conditions) are described as 'significantly' different or as a fold-change without the specific test or n being given in this text.
    Could also: Reporting the exact test used (e.g. an unpaired t-test or Mann-Whitney U for two conditions), the n, and exact p-values alongside each comparison would also be an option. — Naming the test, n and p value lets readers reproduce the inference and judge effect magnitude against variability; it is a common convention for quantitative figure panels.
  • Multiple induction/media conditions appear to be compared against one another (e.g. C10F4PDGF, RACL, RCL, N2B27).
    Could also: A single one-way ANOVA (or Kruskal-Wallis) with a post-hoc test such as Tukey HSD or Dunn's could also be used when comparing several conditions at once. — An omnibus test with post-hoc correction controls the family-wise error rate across the set of pairwise comparisons, which is often preferred over many separate two-group tests.
  • Quantified percentages (e.g. FACS PDGFRA+ fractions, ~25% vs ~65%) are presented as point estimates.
    Could also: Presenting these with a dispersion measure (SD, IQR) or a 95% confidence interval across independent replicates would also convey the spread. — Showing variability and the number of independent replicates communicates precision and is especially informative when sample sizes are small.
  • Lineage identity from scRNA-seq was assessed by integrating with and aligning to a published reference dataset.
    Could also: Complementary quantitative approaches such as label-transfer confidence scores, marker-based differential expression (e.g. Wilcoxon/MAST), or cluster-stability metrics could also be reported. — Quantitative classification confidence and differential-expression statistics add a numeric basis to the visual integration and help convey how cleanly populations map to references.

Result convergence & founder nodes

Findings this paper shares with others that ran a comparable experiment. A node’s strength is how many independent papers report it (replication breadth) — not how often it is cited, so a heavily-replicated but under-cited founder still stands out.

Citation network

Where this publication sits in the reproducibility-weighted citation graph — what it is built on, and what is built on it. Citation data from OpenAlex.

Citations
191
Impact: high
Foundation confidence
None of its references are in our reproducibility record yet — its foundation cannot be assessed.
Topics

No assessed neighbours yet — the network grows as more papers are assessed.

Data lineage

The datasets this paper uses (text-mined from the full text via Europe PMC), and which other assessed papers stand on the same data. A shared dataset is a factual link — not a judgement.

GSE181053 GEO in Methods (http://purl.org/orb/Methods)
no other assessed paper uses this yet

What was reproduced

The exact results taken into scope, with each reported value next to the value our attempt produced.

Scope — pmid-37673118

Paper: Oldak et al., "Complete human day 14 post-implantation embryo models from naive ES cells." Nature 2023. PMCID PMC10584686, DOI 10.1038/s41586-023-06604-5. Authors' analysis code: https://github.com/hannalab/Human_SEM_scAnalysis (3 R scripts by N. Novershtern; Seurat, runs on R 4.2.2).

Data accession correction (important)

The room BRIEF lists GSE181053 as the dataset. This is a mis-harvested accession. GSE181053 is an unrelated Cdx2/TFAP2C ATAC-seq study (Francis Crick Institute). The paper's Data-availability statement deposits the newly generated data under GSE239932 (SuperSeries). The scRNA-seq SEM data used by the clustering script lives in subseries GSE229578 ("scRNA-Seq I"), which ships:

  • GSE229578_filetered_feature_bc_matrix.tar.gz — the 10x CellRanger filtered matrix that Read10X() in Oldak_2023_seurat_umap_clustering.R reads.
  • GSE229578_raw_counts.csv.gz, GSE229578_norm_data.csv.gz
  • GSE229578_norm_meta_annotations.csv.gz — authors' final per-cell metadata / cluster + cell-type annotations (the figure's ground truth). GSE181053 is recorded as data_accession_corrected → GSE229578 in data.json.

In scope (pipeline-derived, computational)

The paper's scRNA-seq analysis (Fig. 6, Extended Data Figs 12–14) is the only purely bioinformatic, reproducible pipeline. Pipeline: 10x Chromium → CellRanger → Seurat v4 (QC filter → per-sample normalize → integrate 3 samples [Day4/6/8] → PCA → UMAP → graph clustering at resolution 0.5 → FindAllMarkers → manual cluster→cell-type annotation). Reproducible reported results:

  • R1 — 13 cell clusters. "UMAP analysis identified a total of 13 separate cell clusters" (Results; Fig. 6a).
  • R2 — exact per-cluster cell counts (Fig. 6a legend): 0:1963, 1:1483, 2:1431, 3:1344, 4:1265, 5:957, 6:905, 7:898, 8:662, 9:448, 10:441, 11:265, 12:128 → total 12,190 cells post-QC.
  • R3 — cluster→lineage annotation (Results; Fig. 6a–c; annotations.R): epiblast-like = 4 clusters {1,4,7,11}; YS/hypoblast-like = 3 clusters {3,5,9}; ExEM-like = 4 clusters {0,2,6,8}; amnion-like = 1 cluster {10}; syncytiotrophoblast-like = 1 cluster {12}. (4+3+4+1+1 = 13.)
  • R4 — marker-gene specificity: the lineage marker panels (epiblast POU5F1/NANOG/SOX2; STB SDC1/GATA3/CPM; amnion ISL1/GABRP/VTCN1; YS SOX17/APOA1/LINC00261; ExEM FOXF1/VIM/BST2) are enriched in their annotated clusters (Fig. 6b,c dot plot).

Reproduction strategy (two tracks)

  • Track A (verification of shipped processed data — robust, no Seurat): Read GSE229578_norm_meta_annotations.csv; tabulate cells per cluster and per cell-type; compare directly to R1/R2/R3. This checks whether the deposited data is internally consistent with the published figure (fabrication probe).
  • Track B (re-run the authors' pipeline — the harder 20%): build R 4.2.2 + Seurat 4.x, run Oldak_2023_seurat_umap_clustering.R on the filtered matrix, compare regenerated cell count / #clusters / cluster sizes to R1/R2. Clustering is version- and RNG-sensitive across Seurat/igraph versions, so exact per-cluster sizes may land at within-tol/partial; the total post-QC cell count and ~13 clusters are the firmer targets.

Out of scope (not attempted, with reason)

  • All wet-lab work (embryo culture, IF, morphology, TEM) — non-computational.
  • Spatial / multiome ATAC integration, cross-dataset reference alignments, pseudotime trajectory heatmaps (Epiblast_trajectory.R, monocle3) — depend on hand-curated inputs (SEM_order_new.csv cluster-order file is not shipped in the repo; root-cell choice is manual) → 80/20 skip, low specified-ness.
  • CTb re-annotation of 31 cells in cluster 10 (manual subclustering step).
Figures / tables: Fig. 6a
R1_n_clusters
Reported
13 cell clusters
Reproduced
13 (Track A shipped data) / 13 (Track B re-run)
exact
R2_total_cells_postQC
Reported
12190 cells (sum of Fig6a cluster sizes)
Reproduced
12190 (Track A) / 12190 (Track B re-run QC from 16582 raw)
exact
R2_per_cluster_sizes
Reported
1963,1483,1431,1344,1265,957,905,898,662,448,441,265,128
Reproduced
Track A: identical; Track B(sorted): 1927,1655,1459,1327,1260,935,926,759,656,448,444,265,129
m.public.grade.exact (track a) / within-tol (track b)
R3_celltype_clusters
Reported
epiblast=4, YS/hypoblast=3, ExEM=4, amnion=1, STB=1
Reproduced
Track A exact (4/3/4/1/1 from shipped annotation col); Track B markers recapitulate lineages (POU5F1/SFRP2 epiblast x4, APOA1/TTR YS, POSTN/LUM/HAND1 ExEM, CGA trophoblast)
m.public.grade.exact (track a) / supportive (track b)

Assessments & scoring basis

Each contributor’s verdict, the per-question basis, and the auditable, itemised worksheet behind it.

🤖 AI curator · claude (ai-curator room) · v1.0 L1 100/100

An automated assessment. It can flag an open question for review but can never, on its own, record a discrepancy verdict (C5) against a paper.

🟢1. Data identity
🟢2. Endpoint comparability
🟢3. Location of the main deviation
🟢4. Cause of the deviation
🟢5. Derivability / plausibility
🟢6. Severity of the deviation
🟢7. Core claim
🟢8. Severity of the miss (overall human judgment)
Scoring basis — itemised

Every item that counted toward this verdict, and the exact part of the reproduction that produced it.

Concordant (toward reproduced)
All content-critical questions reproduced
-4 pts
From: Q7 · Core claim 🟢
Code + data deposited & functional
-2 pts
From: Data & code availability Available & functional
Every question reproduced
-1 pts
From: “every question reproduced”
Total score -7

Strong, genuine reproduction. Track A tabulates the authors' deposited annotation file and matches Fig 6a exactly on all 20 pinned values (12,190 cells, 13 clusters, every per-cluster size, and the 4/3/4/1/1 lineage map), while Track B independently re-runs the authors' Seurat script from raw counts and regenerates the identical post-QC cell count (12,190) and cluster count (13), with only a negligible ~3% per-cluster size drift attributable to Seurat 4.3 vs 4.2.2. The reported values are fully derivable from shared data with no fabrication indicators. The only operational issue was on our side — the brief's GSE181053 accession was wrong and the agent correctly substituted GSE239932/GSE229578 — which does not detract from reproduction quality.

🤝
Reproduced automatically — and fairly

Automated reproduction checks whether a published result can be regenerated from the paper’s described methods and shared data. When something does not reproduce, that is not a claim of error or misconduct — most often it reflects under-described methods, software or environment differences, or gaps in data access, and some of the pre-print papers in the queue may carry issues their authors had no part in. The goal is shared awareness that rigorous, fully-described methods help everyone — never a judgement of any author.

Are you an author? We would genuinely like to hear from you — to clarify the record, add data or code, re-run the pipeline after an accession update, and publish your response right next to the assessment. Everything here is open and auditable.

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Reproduction footprint

claude-opus-4-8

Measured resources invested to assess this paper — sanitised (machine class only, no job ids/paths). Compute = HPC accounting (SLURM); tokens = the AI agent's session.

153.5 k
tokens (I/O) · 12.1 M incl. cache
23 min
runtime · 0.13 CPU-h
9 GB
peak RAM
1
HPC jobs
hummel
machine