Jesse Kreger
Reproducibility track record
1
assessed papers
90/100
mean reproducibility
1
reproduced (C1–C2)
0
flagged
8
total citations
flag rate:
0%
(0/1)
The share of this author’s assessed papers carrying a ⚑ flag. A concentration is a prompt for expert review — never, on its own, a determination about the person.
Authorship role
first author: 1
last author: 0
Topics
Funders
—
Frequent co-authors
Institutions
University of Southern California 1
Geography (author institutions)
US 1
Co-author network
Collaborators, sized by shared output and coloured by their own reproducibility (green = high, red = low). Click a node to open their card. A pattern is a prompt for review, never a determination.
How this author’s assessed papers reproduced — the outcome of reproduction attempts, not a judgement of the person. Coverage is partial and grows over time.
Assessed papers (1)
Complete publication record (60)
Request a reproduction →1 assessed by us (1 reproduced) · 59 not yet assessed — every PubMed paper on record, linked below.
-
A class act: HDAC1- Malat1 regulates MDSC apoptosis and cell cycling to decrease suppression of T cells ↗bioRxiv (Cold Spring Harbor Laboratory) · 2026 · PMID 41929180not yet assessed
-
Abstract PS3-13-16: Cancer systems immunology unravels complexity of reversing immune suppression and predicts beyond RECIST in metastatic breast cancer ↗Clinical Cancer Research · 2026not yet assessed
-
Abstract C058: Myeloid-T cell interactions and B cell activation mediate response to treatment with entinostat and checkpoint inhibitors in metastatic breast cancer ↗Cancer Immunology Research · 2026not yet assessed
-
Immune Circuit Rewiring by HDAC Inhibition Enables Response to Dual Checkpoint Blockade in Metastatic Breast Cancer ↗Cancer Research · 2026 · PMID 42578817not yet assessed
-
Sequencing analysis reveals evidence of immune activation in advanced HER2 negative breast cancer responders treated with entinostat + nivolumab + ipilimumab ↗Research Square · 2025 · PMID 40585251not yet assessed
-
Abstract B020: Dual checkpoint inhibition using anti-PD-1 and anti-CTLA-4 in combination with entinostat increases treatment efficacy and decreases suppressive signaling between myeloid cells and T cells in breast-to-lung metastasis ↗Cancer Immunology Research · 2025not yet assessed
-
Abstract A068: HDAC1 and HDAC3 inhibition decreases myeloid derived suppressor cell function through STAT3-mediated apoptosis ↗Cancer Immunology Research · 2025not yet assessed
-
Cancer systems immunology reveals myeloid—T cell interactions and B cell activation mediate response to checkpoint inhibition in metastatic breast cancer ↗bioRxiv (Cold Spring Harbor Laboratory) · 2025 · PMID 40661534not yet assessed
-
Abstract P1-04-21: Suppressing suppression: histone deacetylase inhibition primes the metastatic tumor microenvironment to respond to checkpoint inhibition by dynamic rewiring of cell interactions ↗Clinical Cancer Research · 2025not yet assessed
-
Beyond RECIST: mathematical modeling and Bayesian inference reveal the importance of immune suppressive parameters in metastatic breast cancer ↗bioRxiv (Cold Spring Harbor Laboratory) · 2025 · PMID 40832320not yet assessed
-
735 T cell interactions and B cell activation mediate response to treatment with entinostat and checkpoint inhibitors in metastatic breast cancer ↗Regular and Young Investigator Award Abstracts · 2025not yet assessed
-
not yet assessed
-
not yet assessed
-
Developmental hematopoietic stem cell variation explains clonal hematopoiesis later in lifeNature Communications · 2024 · PMID 39592593L1 90/100
-
Gastrulation-stage gene expression in Nipbl +/− mouse embryos foreshadows the development of syndromic birth defects ↗Science Advances · 2024 · PMID 38507484not yet assessed
-
A new Neu—a syngeneic model of spontaneously metastatic HER2-positive breast cancer ↗Clinical & Experimental Metastasis · 2024 · PMID 38717519not yet assessed
-
Developmental hematopoietic stem cell variation explains clonal hematopoiesis later in life ↗bioRxiv (Cold Spring Harbor Laboratory) · 2024 · PMID 38496542not yet assessed
-
Intratumoral CXCL12 Gradients Contextualize Tumor Cell Invasion, Migration and Immune Suppression in Breast Cancer ↗bioRxiv (Cold Spring Harbor Laboratory) · 2024 · PMID 39464015not yet assessed
-
Mimicking the breast metastatic microenvironment: characterization of a novel syngeneic model of HER2 + breast cancer ↗bioRxiv (Cold Spring Harbor Laboratory) · 2024 · PMID 38352476not yet assessed
-
Abstract 6522: Epigenetic modulation decreases myeloid suppression to improve response to immune checkpoint inhibition in breast cancer ↗Cancer Research · 2024not yet assessed
-
not yet assessed
-
not yet assessed
-
not yet assessed
-
not yet assessed
-
not yet assessed
-
not yet assessed
-
not yet assessed
-
770 Entinostat attenuates myeloid cell immunosuppression and increases the efficacy of checkpoint inhibition in a HER2+ mouse model of breast-to-lung metastasis ↗Regular and Young Investigator Award Abstracts · 2024not yet assessed
-
874 Epigenetic modulation of myeloid derived suppressor cells decreases suppressive signaling through the STAT3 pathway ↗Regular and Young Investigator Award Abstracts · 2024not yet assessed
-
Myeloid-Derived Suppressor–Cell Dynamics Control Outcomes in the Metastatic Niche ↗Cancer Immunology Research · 2023 · PMID 36848523not yet assessed
-
Gastrulation-stage gene expression in Nipbl +/- mouse embryos foreshadows the development of syndromic birth defects ↗bioRxiv (Cold Spring Harbor Laboratory) · 2023 · PMID 37905011not yet assessed
-
Abstract 5167: Entinostat's modulation of myeloid derived suppressor cells through the STAT3-NFκB-AP-1 axis decreases suppressive signaling ↗Cancer Research · 2023not yet assessed
-
Abstract 3256: Entinostat enhances the efficacy of checkpoint inhibition in breast-to-lung metastases and is associated with alterations in the phenotype and function of myeloid cell populations ↗Cancer Research · 2023not yet assessed
-
not yet assessed
-
not yet assessed
-
not yet assessed
-
not yet assessed
-
not yet assessed
-
not yet assessed
-
not yet assessed
-
not yet assessed
-
not yet assessed
-
not yet assessed
-
not yet assessed
-
The role of migration in mutant dynamics in fragmented populations ↗Journal of Evolutionary Biology · 2022 · PMID 36514852not yet assessed
-
Myeloid-derived suppressor cell dynamics control outcomes in the metastatic niche ↗bioRxiv (Cold Spring Harbor Laboratory) · 2022not yet assessed
-
not yet assessed
-
not yet assessed
-
not yet assessed
-
A hybrid stochastic-deterministic approach to explore multiple infection and evolution in HIV ↗PLoS Computational Biology · 2021 · PMID 34936647not yet assessed
-
Quantifying the dynamics of viral recombination during free virus and cell-to-cell transmission in HIV-1 infection ↗Virus Evolution · 2021 · PMID 34012557not yet assessed
-
The role of migration in mutant evolution in fragmented populations ↗bioRxiv (Cold Spring Harbor Laboratory) · 2021not yet assessed
-
The effect of multiple infection, recombination, and synaptic transmission on the evolution of human immunodeficiency virus infectioneScholarship (California Digital Library) · 2021not yet assessed
-
Effect of synaptic cell-to-cell transmission and recombination on the evolution of double mutants in HIV ↗Journal of The Royal Society Interface · 2020 · PMID 32208824not yet assessed
-
Quantifying the dynamics of viral recombination during free virus and cell-to-cell transmission in HIV-1 infection ↗bioRxiv (Cold Spring Harbor Laboratory) · 2020not yet assessed
-
A hybrid stochastic-deterministic approach to explore multiple infection and evolution in HIV ↗bioRxiv (Cold Spring Harbor Laboratory) · 2020not yet assessed
-
Fortran Sample Code from Effect of synaptic cell-to-cell transmission and recombination on the evolution of double mutants in HIV ↗Figshare · 2020not yet assessed
-
C Sample code from Effect of synaptic cell-to-cell transmission and recombination on the evolution of double mutants in HIV ↗Figshare · 2020not yet assessed
-
Effect of synaptic cell-to-cell transmission and recombination on the evolution of double mutants in HIV ↗bioRxiv (Cold Spring Harbor Laboratory) · 2019not yet assessed
-
not yet assessed
Full bibliography from OpenAlex; reproducibility verdicts matched by PMID.
Author attribution follows OpenAlex disambiguation, which is imperfect — a researcher's papers can be split across profiles or mixed with a namesake.
Merged across profiles sharing this ORCID where present. See every “Kreger J” paper on PubMed ↗